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The acetylation status of NF-kB p65 and the complex with nuclear factor kappa-light-chain-enhancer of activated B cells NF-B (NF-B) p65 and STAT1 suppressed the transcriptional activity of NF-kB p65 and attenuated the central inflammatory response mediated by microglia and led to the neuroprotective effect of VPA therapeutic metabolites [109]

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